occlusion

enviro, behavioral, bio factor ASSOCIATED with dz

biologically plausible as causal agent for dz
or
precede devlopment of dz

operates in harmony
no pathologic manifestation in supporting structures of teeth
stresses on teeth are dissipated normally
balance between stress ~ adaptive capacity

occlusion without harmony
DOES present pathology in supporting structures
stress NOT dissipated normally
NO balance between stress and adaptivity

EXCESSIVE occlusal force

movement about fulcrum
zone of pressure: OPPOSES xs force, on same side
zone of tension: TOWARDS xs force, on opp side

BV enlarge, PDL disorganize (thus widen), collagen destroyed
osteoclast predominate
pressure zone: alv bone resorption
tooth mobility

new bone formed between teeth and at B/L plates

widening of socket and PDL > allow movt of tooth away from traumatic force
mobility remain constant, not progressive (can only be progressive if occlusal forces > adaptive nature)

NO! but they can contribute to the progression of perio dz
thus it is a local risk factor

initiating inflammation or pocket formation in absence of plaque
does not increase gingivitis

existing pdtitis with xs occlusal forces, when inflam reaches perio membrane fibers it spread further upon occlusal force

palpable or visible movt of a tooth upon occlusal forces

teeth move anteriorly and rotate

tissue changes from xs occlusal forces on NORMAL SUPPORT teeth
occurs in presence of normal bone and att levels, but xs occl force
(basically no existing pdtitis)

tissue changes from xs occl forces on REDUCED SUPPORT teeth
occur in presence of bone and att loss

centric occlusion
smooth excursive without interferences

coronoplasty

attrition (no att loss)
perio therapy

decrease mobility

defer until:
marginal inflam is controlled
time is allowed for tissue healing
when residual mobility is from adverse loading, NOT DECREASED SUP