2Angina types
-Stable: predictable & constant
-Unstable: pain that doesn’t go away with rest; can lead to a heart attack
3Drugs of choice to treat angina:
-nitroglycerin: vasodilator (side effect: hypotension)
4What is initated first at the time of a heart issue?
Oxygen therapy
5Nurse’s duty in order to differentiate type of angina
Obtain a detailed patient history
6Cardiomyopathy
disease of the heart muscle
7Types of cardiomyopathies
Dilated & Hypertrophic
8Dilated Cardiomyopathy
(the most common) a significant dilation of ventricules without simultaneous hypertrophic and systolic dysfunction)
9Hypertrophic Cardiomyopathy
(common in athletes) it is genetic; leads to increase heart muscle size and mass
10First two things to keep in mind when dealing with cardiomyopathy
Sodium Levels & Fluid Overload
11DVT
immobile people are the most at risk (bedridden, surgical pts, etc); can travel and cause a PE; treated with heparin & warfarin
12DVT labs
INR (therapeutic levels on warfarin: 2.0-3.0; normal therapeutic range: 2-3.5)
13thromoboflabitis
doesn’t require hospitalization; rest, wear compression stockings
14Dysrhythmia
focus on EKG results
15P-Wave
represents the electrical impulse of the upper chambers of the heart
16QRS
represents the depolarization of the lower chambers of the heart
17T-waves
represents the depolarization of the ventricles (return to electrical rest)
18PE
the obstruction of the pulmonary artery or one of its branches by a thrombus (or thrombi) that originates somewhere in the venous system or in the right side of the heart
19Clinical manifestations of PE
chest pain, shortness of breath (dyspnea)
20HOW to manage PE
Full assessment; administer O2
21Implantable cardioverter defibrillator (ICD)
is an electronic device that detects and terminates life-threatening episodes of tachycardia or fibrillation, especially those that are ventricular in origin
22ACS
acute coronary syndrome: is an emergent situation characterized by an acute onset of myocardial ischemia that results in myocardial death
23Labs associated with MI
EKG, ECG, Troponin, Creatine Kinase, Myoglobin
24Patient’s risk while getting a stent placed
Bleeding
25How long is a patient admitted for following a valvuoplasty/valve replacement procedure?
24-28 hours
26Normal Values
-pH: 7.35-7.45
-PCO2: 35-45mm Hg
-PO2: >80mm Hg
-HCO3: 22-26 mEq/L
-SaO2 %: >94%
27Pneumothorax
occurs when the parietal or visceral pleura is breached and the pleural space is exposed to positive atmospheric pressure
28Types of Pneuothorax
-Simple
-Traumatic
-Tension
29Simple Pneuothorax
spontaneous; occurs when air enters the pleural space through a breach of either the parietal or visceral pleura. Most commonly, this occurs as air enters the pleural space through the rupture of a bleb or a bronchopleural fistula. A spontaneous pneumothorax may occur in an apparently healthy person in the absence of trauma due to rupture of an air-filled bleb, or blister, on the surface of the lung, allowing air from the airways to enter the pleural cavity
30Traumatic Pneumothorax
occurs when air escapes from a laceration in the lung itself and enters the pleural space or from a wound in the chest wall
31Traumatic Pneumothorax
occurs when air is drawn into the pleural space from a lacerated lung or through a small opening or wound in the chest wall; causes the lungs to collapse and the heart, great vessels and trachea to shift towards the unaffected side of the chest(aka mediastinal shift)
32Clinical manifestations of a Pneumothorax
pain is usually sudden; the trachea is midline, expansion of the chest is decreased, breath sounds may be diminished or absent, and percussion of the chest may reveal normal sounds or hyperresonance depending on the size of the pneumothorax. In a tension pneumothorax, the trachea is shifted away from the affected side, chest expansion may be decreased or fixed in a hyperexpansion state, breath sounds are diminished or absent, and percussion to the affected side is hyperresonant
33SARS Signs & symptoms
non-productive cough, sore throat, fatigue, muscle aches, congestion, nausea
34Metabolic Acidosis
is a common clinical disturbance characterized by a low pH due to an increased H+ concentration and a low plasma bicarbonate concentration.
35High anion gap metabolic acidosis
occurs when there is an excessive accumulation of acids. High anion gap occurs in lactic acidosis, salicylate poisoning (acetylsalicylic acid), renal failure, methanol, ethylene or propylene glycol toxicity, DKA, and ketoacidosis that occurs with starvation. The high amount of hydrogen ions due to the acids present are neutralized and buffered by HCO3− causing the bicarbonate concentration to fall and become exhausted
36Metabolic Alkalosis is often caused by
severe vomiting or gastric suctioning
37Nursing responsibilities for MA
correcting underlying acid-base disorder; restoring normal fluid volume by administering NaCl
38DKA
caused by an absence or markedly inadequate amount of insulin; glucose levels: 250-800; signs/symptoms: fruity breath and Kussmaul respirations; major concern: POTASSIUM (affecting the heart); monitor blood sugar
39Three main clinical features of DKA
-Hyperglycemia
-Dehydration & electrolyte loss
-Acidosis
40HHS
is a metabolic disorder most often of type 2 diabetes resulting from a relative insulin deficiency initiated by an illness that raises the demand for insulin. This is a serious condition in which hyperosmolality and hyperglycemia predominate, with alterations of the sensorium (sense of awareness)
41Clinical manifestations of HHS
hypotension, profound dehydration (dry mucous membranes, poor skin turgor), tachycardia, and variable neurologic signs (e.g., alteration of consciousness, seizures, hemiparesis
42Treatment of HHS
fluid replacement; correction of electrolyte imbalances; insulin administration
43Viral Hepatitis
-Hep A
-Hep B
-Hep C
44Hep B
transmitted primarily through blood, prevented by: safe sex, avoid high risk behaviors, hygiene
45Hep C
transmitted by blood transfusion and sexual contact
46Hep C treatment
Protease Inhibitor Therapies for 8-12 weeks of oral therapy